The Bright Insight Blog

For many years, AuDHDers may have been misidentified as having bipolar disorder, meaning that studies of bipolar populations could actually be describing this experience. This raises critical questions about how much of the literature reflects autistic and dynamically attentive lived realities rather than distinct mood disorder trajectories.

Looking across the lifespan, hyperneuroplasticity emerges as a systemic orientation that permeates every stage of development, rather than a temporary trait to be gained or lost.

For humans navigating giftedness, neurodivergence, or trauma recovery, the octopus serves as both metaphor and scientific model. It reminds us of our capacity for deep, systemic change and calls us to radically accept a fluidity that can be both barrier and brilliance.

Framing hyperneuroplasticity through epigenetics grounds this construct in biology and emphasizes both its risks and its extraordinary potential.

The DecodeME study marks a turning point. It acts as a compass pointing us to a terrain where ME/CFS is genetic, biological, and systemic, rather than irrational or hyperbolic. Hyperneuroplasticity tells us that this terrain is shared with those navigating MCAS, EDS, autism, ADHD, trauma, giftedness, and beyond.

Taken together, hyperneuroplasticity, the blood–brain barrier, EDS, MCAS, dysautonomia, and FND form a constellation of overlapping expressions of a body and brain that are open, sensitive, and dynamic. This reframing points toward interventions that aim to stabilize barriers, modulate immune activity, regulate autonomic flow, and most importantly, harness hyperneuroplasticity constructively through learning, creativity, and self-directed adaptation.